RESUMO
The aim of this case study was to evaluate the clinical and productive data of Nellore cows during pregnancy and lactation, supplemented or not with P. Ninety-five pregnant heifers were divided into two groups (G40 and G0). Until the date of calving animals from G40 received a mineral mixture composed of 224g of Na and 40g of P/kg, and G0 received only NaCl. After calving heifers were divided into four treatments: from G40, 28 first-calf cows continued to receive the mineral mixture containing 224g of Na and 40g of P/kg (group 40/40) and 12 started to receive only NaCl (group 40/0); from G0, 26 continued to receive only NaCl (group 0/0) and 15 started to receive a mixture containing 224g of Na and 40g of P/kg (group 0/40). In the second experimental phase, 205 multiparous cows were divided into two groups: G1 (40/40) consisting of 40 cows, receiving a mineral mixture containing 244g of Na and 40g/P per kg, during pregnancy and lactation, and group 2 (0/0) formed by 165 cows, which were supplemented only with NaCl. In both experiments, whether in pregnant or lactating breeders, there was not any sign of P deficiency or differences in calves LW at 120 and 210 days.
Este estudo avaliou os aspectos clínicos e produtivos de matrizes Nelore, suplementadas ou não com P. Noventa, e cinco nulíparas prenhes foram distribuídas em dois grupos (G40 e G0). Os animais do G40 receberam uma mistura mineral com 224g de Na e 40g de P/kg, até a data do parto, e o G0 recebeu apenas NaCl. Após o parto, as primíparas foram divididas em quatro grupos: do G40, 28 continuaram a receber mistura contendo 224g de Na e 40g de P/kg (grupo 40/40), e 12 passaram a receber apenas NaCl (grupo 40/0); do G0 26 continuaram recebendo apenas NaCl (grupo 0/0), e 15 passaram a ser suplementadas com a mistura contendo 224g de Na e 40g de P/kg (grupo 0/40). Na segunda fase experimental, 205 vacas foram divididas em dois grupos: G1 (40/40), composto por 40 vacas, suplementadas com a mistura mineral contendo 244g de Na e 40g de P/kg, durante a gestação e a lactação, e o G2 (0/0) foi formado por 165 vacas, as quais foram suplementadas apenas com NaCl, durante a gestação e a lactação. Em ambos os experimentos, seja nas matrizes em gestação ou naquelas em lactação, não houve qualquer sinal da deficiência de P, e isso se refletiu na ausência de diferenças nos pesos dos bezerros aos 120 e 210 dias.
Assuntos
Animais , Feminino , Gravidez , Bovinos , Peso ao Nascer , Fósforo na Dieta/administração & dosagem , Fósforo na Dieta/análiseRESUMO
Conidiobolomycosis and pythiosis are important diseases of sheep in midwestern Brazil. Veterinary practitioners consider it difficult to differentiate between these diseases because they have similar clinical features. In this study, 186 sheep were subjected to necropsy examination over a 6-year period. Thirty (16.1%) cases of rhinitis in sheep that were caused by Conidiobolus lamprauges (n = 15) or Pythium insidiosum (n = 15) were investigated further. The lesions of C. lamprauges infection were mainly rhinopharyngeal (86.7%), localized to the ethmoidal region and associated with exophthalmos. The lesions appear as a white to yellow, firm mass that microscopically appears as a granulomatous inflammatory reaction with numerous giant cells. In contrast, P. insidiosum infection is associated with rhinofacial (93.3%) lesions that mainly involve the frontal region and hard palate and appear as an irregular, friable, yellow to red mass. Microscopically, pythiosis presents as diffuse necrotizing eosinophilic rhinitis. Immunohistochemistry using polyclonal antisera raised specifically against the two organisms was used to confirm the identity of the infectious agent in each disease. This study reports the first immunohistochemical diagnosis of conidiobolomycosis and the first description of a rhinopharyngeal lesion caused by P. insidiosum in sheep.
Assuntos
Pitiose/veterinária , Infecções Respiratórias/patologia , Rinite/patologia , Doenças dos Ovinos/patologia , Zigomicose/veterinária , Animais , Conidiobolus , Imuno-Histoquímica , Pitiose/patologia , Pythium , Infecções Respiratórias/microbiologia , Rinite/microbiologia , Ovinos , Doenças dos Ovinos/microbiologia , Carneiro Doméstico , Zigomicose/patologiaRESUMO
Relata-se necrose hepatocelular em suínos após consumo de ração que continha grãos de sorgo-granífero (Sorghum bicolor) acidentalmente contaminado com sementes de Crotalaria spectabilis. Morreram 76 suínos em quatro propriedades no município de Juscimeira, MT. Os sinais clínicos iniciaram-se 24-48 horas após o consumo da ração contaminada e foram caracterizados por depressão, letargia, apatia, inapetência, vômito, mucosas ictéricas ou pálidas, ascite, decúbito esternal, decúbito lateral com movimentos de pedalagem e convulsões, a evolução clínica foi de 48-60 horas seguida de morte. As Principais alterações macroscópicas foram fígado aumentado de tamanho com evidenciação do padrão lobular, ascite e hidrotórax com líquido de coloração amarelo avermelhado contendo filamentos com aspecto de fibrina, linfonodos aumentados e edema pulmonar interlobular. A doença foi reproduzida utilizando-se 16 suínos divididos em seis grupos que receberam sementes de C. spectabilis em diferentes doses. Necrose hepatocelular ocorreu em sete suínos, sendo dois que receberam doses diárias 2,5g/kg e cinco que receberam doses únicas de 5,0 e 9,5g/kg. Dez doses diárias de 0,5 e 1,25g/kg causaram fibrose hepática.(AU)
Hepatocellular necrosis are reported in swine after consumption of diets containing grains of "sorgo-granífero" (Sorghum bicolor) accidentally contaminated with Crotalaria spectabilis seeds in the municipality of Juscimeira, MT. Clinical signs began 24-48 hours after consumption of contaminated ration and were characterized by depression, lethargy, apathy, loss of appetite, vomiting, pale or jaundiced mucous membranes, ascites, lateral recumbency, the lateral position with paddling and convulsions, clinical outcome was 48-60 hours followed by death. 76 pigs died in four properties. The main gross lesions were liver increased in size and lobular illustration with red-brown central areas interspersed with yellowish areas, ascites and hydrothorax with reddish-yellow liquid containing filaments with aspect of fibrin, enlarged lymph nodes and interlobular pulmonary edema. The disease was experimentally reproduced with 16 pigs divided into six groups that received seeds of C. spectabilis in different doses. Hepatocellular necrosis occurred in seven pigs, two of which received daily doses of 2.5g/kg and five who received single doses of 5 and 9.5g/ kg. Ten daily doses of 0.5 and 1.25g/kg caused liver fibrosis.(AU)
Assuntos
Animais , Crotalaria/toxicidade , Necrose Hepática Massiva/veterinária , Plantas Tóxicas/intoxicaçãoRESUMO
Relata-se necrose hepatocelular em suínos após consumo de ração que continha grãos de sorgo-granífero (Sorghum bicolor) acidentalmente contaminado com sementes de Crotalaria spectabilis. Morreram 76 suínos em quatro propriedades no município de Juscimeira, MT. Os sinais clínicos iniciaram-se 24-48 horas após o consumo da ração contaminada e foram caracterizados por depressão, letargia, apatia, inapetência, vômito, mucosas ictéricas ou pálidas, ascite, decúbito esternal, decúbito lateral com movimentos de pedalagem e convulsões, a evolução clínica foi de 48-60 horas seguida de morte. As Principais alterações macroscópicas foram fígado aumentado de tamanho com evidenciação do padrão lobular, ascite e hidrotórax com líquido de coloração amarelo avermelhado contendo filamentos com aspecto de fibrina, linfonodos aumentados e edema pulmonar interlobular. A doença foi reproduzida utilizando-se 16 suínos divididos em seis grupos que receberam sementes de C. spectabilis em diferentes doses. Necrose hepatocelular ocorreu em sete suínos, sendo dois que receberam doses diárias 2,5g/kg e cinco que receberam doses únicas de 5,0 e 9,5g/kg. Dez doses diárias de 0,5 e 1,25g/kg causaram fibrose hepática.
Hepatocellular necrosis are reported in swine after consumption of diets containing grains of "sorgo-granífero" (Sorghum bicolor) accidentally contaminated with Crotalaria spectabilis seeds in the municipality of Juscimeira, MT. Clinical signs began 24-48 hours after consumption of contaminated ration and were characterized by depression, lethargy, apathy, loss of appetite, vomiting, pale or jaundiced mucous membranes, ascites, lateral recumbency, the lateral position with paddling and convulsions, clinical outcome was 48-60 hours followed by death. 76 pigs died in four properties. The main gross lesions were liver increased in size and lobular illustration with red-brown central areas interspersed with yellowish areas, ascites and hydrothorax with reddish-yellow liquid containing filaments with aspect of fibrin, enlarged lymph nodes and interlobular pulmonary edema. The disease was experimentally reproduced with 16 pigs divided into six groups that received seeds of C. spectabilis in different doses. Hepatocellular necrosis occurred in seven pigs, two of which received daily doses of 2.5g/kg and five who received single doses of 5 and 9.5g/ kg. Ten daily doses of 0.5 and 1.25g/kg caused liver fibrosis.