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The toxin biliatresone causes mouse extrahepatic cholangiocyte damage and fibrosis through decreased glutathione and SOX17.
Waisbourd-Zinman, Orith; Koh, Hong; Tsai, Shannon; Lavrut, Pierre-Marie; Dang, Christine; Zhao, Xiao; Pack, Michael; Cave, Jeff; Hawes, Mark; Koo, Kyung A; Porter, John R; Wells, Rebecca G.
Afiliação
  • Waisbourd-Zinman O; Division of Gastroenterology, Hepatology and Nutrition, The Children's Hospital of Philadelphia, Philadelphia, PA.
  • Koh H; Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA.
  • Tsai S; Department of Pediatrics, Yonsei University College of Medicine, Severance Children's Hospital, Seoul, South Korea.
  • Lavrut PM; Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA.
  • Dang C; Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA.
  • Zhao X; Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA.
  • Pack M; Department of Biological Sciences, University of the Sciences, Philadelphia, PA.
  • Cave J; Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA.
  • Hawes M; Division of Gastroenterology, Department of Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA.
  • Koo KA; Department of Economic Development, Jobs, Transport and Resources, Government of Victoria, Victoria, Australia.
  • Porter JR; Department of Economic Development, Jobs, Transport and Resources, Government of Victoria, Victoria, Australia.
  • Wells RG; Department of Biological Sciences, University of the Sciences, Philadelphia, PA.
Hepatology ; 64(3): 880-93, 2016 09.
Article em En | MEDLINE | ID: mdl-27081925
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Atresia Biliar / Ductos Biliares Extra-Hepáticos / Proteínas HMGB / Benzodioxóis / Fatores de Transcrição SOXF / Glutationa Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Revista: Hepatology Ano de publicação: 2016 Tipo de documento: Article País de afiliação: Panamá País de publicação: Estados Unidos
Texto completo
Adicionar na Minha BVS
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Atresia Biliar / Ductos Biliares Extra-Hepáticos / Proteínas HMGB / Benzodioxóis / Fatores de Transcrição SOXF / Glutationa Tipo de estudo: Etiology_studies / Prognostic_studies Limite: Animals Idioma: En Revista: Hepatology Ano de publicação: 2016 Tipo de documento: Article País de afiliação: Panamá País de publicação: Estados Unidos